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I. CLINICAL OBSERVATIONS

JOHN A. TOOMEY, M.D.

Am J Dis Child. 1936;51(1):58-68.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

The production of the spinal type of lesion usually seen in human beings with poliomyelitis can be best explained by assuming that the factor that causes the disease enters through the gastro-intestinal tract and from there spreads to the spinal cord by way of the sympathetic nervous system.¹ The production of the bulbar type of the disease may be explained by assuming that the virus can also spread from the gastro-intestinal tract to the medullary area by way of the axis-cylinders of both vagus nerves.² Symptoms of illness become evident as soon as the nucleus ambiguus is involved. The other cranial nerves, or components, especially the nuclei of the third, sixth, ninth and eleventh nerves situated in the medullary area, may also become involved, but only secondarily. Occasionally the nucleus of the seventh nerve becomes paralyzed along with the other nuclei. There are a few cases of the . . . [Full Text PDF of this Article]

Author Affiliations
CLEVELAND

From the Department of Pediatrics, Western Reserve University, and the Division of Contagious Diseases, the City Hospital.

Footnotes
The expenses for the experimental work reported in part II were defrayed in part by a grant from the Marion R. Spellman Fund, the Cleveland Foundation.

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