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REGINALD A. HIGGONS, M.D.

Am J Dis Child. 1935;50(1):162-165.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

There is considerable literature on the subject of hypoglycemia in adults, both with and without insulin therapy. Very little, however, has been written concerning the state of hypoglycemia as a cause of symptoms in new-born infants. The effect of hypoglycemia in the production of neurologic symptoms in adults has been well described by Banting and his coworkers.¹

Britton² noted that the blood sugar of new-born kittens was usually maintained at a normal level even though the mother's blood sugar was severely reduced by insulin or greatly increased by epinephrine or dextrose. Bowen and Heilbrun,³ however, reported instances in which the fetus of a diabetic mother had hypertrophic islands of Langerhans in the pancreas.

In the literature on diabetes during pregnancy very little is said concerning the sugar metabolism of the child after birth. Stander and Peckham⁴ have expressed the opinion that the excessive size of many . . . [Full Text PDF of this Article]

Author Affiliations
PORT CHESTER, N. Y.

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