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VI. THE PATHOLOGIC PROCESS IN THIRTY-FOUR CASES

GEORGE L. WALDBOTT, M.D.

Am J Dis Child. 1934;47(1):41-60.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

Most recent contributors to the literature on so-called "thymic death" are at a loss to explain this phenomenon. Painstaking research during recent years has led to only a few positive observations. The work of the English Status Lymphaticus Investigation Committee¹ on over 600 autopsies tends to question the existence of a thymic diathesis. The diagnosis of thymic death is frequently made the dumping ground of unexplained sudden death (Kennedy and New²). Enlargement of the thymus is often diagnosed erroneously on insufficient roentgenographic evidence (Hasley and Di Tomasi³). Boyd⁴ observed that a large thymus is rather the rule than the exception in patients dying suddenly. The formerly prevalent conception that pressure on the thymus is responsible for so-called "thymic" asthma, and even for death, has lost considerable ground within recent years.

In spite of these various observations tending to deny the original conception of Paltauf that a . . . [Full Text PDF of this Article]

Author Affiliations
DETROIT

The following pathologists cooperated with me in this study: Dr. C. V. Weller, University Hospital, Ann Arbor (8 cases); Dr. A. M. Young and Dr. B. S. Kline, Mount Sinai Hospital, Cleveland (7 cases); Dr. W. H. Chase, McGill University, Montreal (5 cases); Dr. L. J. Rhea, Montreal General Hospital (3 cases); Dr. P. F. Morse, Harper Hospital, Detroit (3 cases); Dr. A. C. Edwards, Children's Hospital, Detroit (2 cases); Dr. B. Steinberg, Toledo Hospital (2 cases); Dr. M. A. Southwick, University of Chicago (2 cases); Dr. W. H. Brosius, St. Joseph's Mercy Hospital, Detroit (1 case); Dr. J. A. Kasper, Herman Kiefer Hospital, Detroit (1 case).

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