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ARVID WALLGREN, M.D.

Am J Dis Child. 1929;38(4):829-833.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

Acute serofibrinous pleurisy is now almost invariably regarded as due to tuberculosis. In what manner tuberculosis gives rise to it, is, on the other hand, somewhat obscure. As a rule, one has been content to state that the tuberculous infection in some manner produces the exudation. The most obvious and common explanation is that a subpleural tuberculous focus has provoked an irritation of the pleura.

That a local tuberculous focus in the lung or the bronchial glands constitutes an important factor in the genesis of pleurisy is indicated by the fact that a focus of this kind can be detected on the side on which the exudation subsequently arises. If an opportunity presents itself of examining a child a short time before it contracts pleurisy, there are generally found homolateral hilar changes or a homolateral primary focus. Of fifty children examined before the development of pleurisy, forty-eight showed homolateral changes . . . [Full Text PDF of this Article]

Author Affiliations
GOTHENBURG, SWEDEN

From the Medical Department of the Children's Hospital.

Footnotes
Submitted for publication, July 10, 1929.

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