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inasi Özsoylu, MD
Department of Pediatric Hematology Hacettepe University Faculty of Medicine Hacettepe Children's Hospital 06100 Ankara, Turkey

Arch Pediatr Adolesc Med. 1994;148(12):1349.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

I read with interest the article by Grosheide et al¹ published in the December 1993 issue of AJDC.

Six (8.3%) of the 72 infants in their study became positive for hepatitis B surface antigen (HBsAg) despite receiving HB immune globulin at birth and at later active immunization (four times). In addition, eight (11.1%) of the 72 infants became anti–hepatitis B core antigen (HBc) positive at age 12 months or older, which is evidence of HB virus infection. When HBsAg and anti-HBc positivities are combined, more than 19% of the children became infected. More important, despite passive and active immunization, 11 (35.5%) of the 31 infants of Mediterranean origin and three (12.5%) of the 24 infants of Asian origin (25% infection rate for both groups) became infected, these being areas of the world where prevention of infection takes on even more importance.

I wish that the authors also could have . . . [Full Text PDF of this Article]

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