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PANKAJA S. VENKATARAMAN, MD
Department of Pediatrics University of Oklahoma Oklahoma City, OK 73190

REGINALD C. TSANG, MD
Department of Pediatrics and Obstetrics and Gynecology University of Cincinnati Cincinnati, OH 45267

Am J Dis Child. 1985;139(12):1182.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

In Reply.—The questions of Dr Cunningham are important ones and precisely the reasons for the publication of our findings.¹ As Dr Cunningham astutely observes, the serum parathyroid hormone concentrations were also increased in the formula-fed infants without symptoms, at least in reference to human-milk—fed infants. The report that tetany in infants fed older formulas was followed by enamel hypoplasia in deciduous teeth actually involved a formula with a higher phosphate content relative to the calcium content of that used in our study. Hyperparathyroidism, indeed, might adversely affect the bones and teeth of formula-fed infants. However, the degree of relative elevation of serum parathyroid hormone concentrations achieved in these infants may not be sufficient to cause any lasting ill effect. These are important questions and extremely interesting ones that we are presently pursuing and we hope to report these results in the near future.

Indeed, bottle-fed infants also have . . . [Full Text PDF of this Article]

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