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JAMES B. HANSHAW, MD

Am J Dis Child. 1982;136(10):886-887.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

In 1964, Medearis observed an association between congenital cytomegalovirus (CMV) infection and sensorineural deafness.¹ That study involved symptomatic newborns, who constitute a modest 6% of the estimated 33,000 infants born with congenital CMV infection in the United States each year. It is now apparent that asymptomatic neonates also have long-term risks and that hearing loss is the most common irreversible sequela of congenital CMV infection.^2,3 The probability of hearing disability is approximately 30% in symptomatic⁴ and 13% in the more common asymptomatic infants with congenital infection.^2,3

In this issue of the Journal (see p 896), Saigal et al report the results of a study involving 15,212 consecutive births in Hamilton, Ontario. They recovered CMV from the urine of 64 infants. Of these, they were able to do audiologic assessments of 41 CMV-infected children as well as 44 matched control subjects. Sensorineural hearing impairment was found in . . . [Full Text PDF of this Article]

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