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Critical Tricuspid Insufficiency due to Papillary Muscle RuptureA Result of Prenatal Hypoxic Insult
Arie L. Alkalay, MD;
David A. Ferry, MD;
Samuel H. Pepkowitz, MD;
Paula J. Chou, MD;
Gary K. Oakes, MD;
Jeffrey J. Pomerance, MD, MPH
Am J Dis Child. 1988;142(7):753-755.
Abstract
Fatal tricuspid insufficiency secondary to papillary muscle rupture due to prenatal hypoxic insult occurred in a full-term newborn. The diagnosis of flail tricuspid valve should be considered when fetal distress is encountered in a newborn with persistent hypoxemia. Prenatal diagnosis of this condition combined with prompt delivery, prostaglandin E1 therapy, and possible surgical repair of the tricuspid valve may improve chances of survival.
(AJDC 1988;142:753-755)
Author Affiliations
From the Division of Neonatology (Drs Alkalay, Chou, and Pomerance) and Cardiology (Dr Ferry), Department of Pediatrics, and the Departments of Pathology and Laboratory Medicine (Dr Pepkowitz) and Obstetrics and Gynecology (Dr Oakes), University of California School of Medicine, Cedars-Sinai Medical Center, Los Angeles.
Footnotes
Accepted for publication Feb 3, 1988.
Reprint requests to Department of Pediatrics, Division of Neonatology, Cedars-Sinai Medical Center, 8700 Beverly Blvd, Los Angeles, CA 90048 (Dr Alkalay).
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