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Gertrude Costin, MD; Maurice D. Kogut, MD; Carol B. Hyman, MD; Jorge A. Ortega, MD

Gertrude Costin, MD; Maurice D. Kogut, MD; Carol B. Hyman, MD; Jorge A. Ortega, MD

Am J Dis Child. 1979;133(5):497-502.

Abstract
• Endocrine function evaluations in 16 patients with β-thalassemia indicate that hypogonadotropic hypogonadism, hypoparathyroidism, and reduced adrenocorticotropic hormone reserve occur frequently, whereas reduced growth hormone and thyroid reserve are less common manifestations. Hemosiderosis-induced damage of the endocrine glands seems to be the main cause for endocrine dysfunction in the patients studied.

(Am J Dis Child 133:497-502, 1979)

Author Affiliations
From the Divisions of Endocrinology and Metabolism (Drs Costin and Kogut) and Hematology-Oncology (Drs Hyman and Ortega), Children's Hospital of Los Angeles; and the Department of Pediatrics (Drs Costin, Kogut, Hyman, and Ortega), University of Southern California School of Medicine, Los Angeles.

Footnotes
Reprint requests to 4650 Sunset Blvd, Los Angeles, CA 90027 (Dr Costin).

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