You are seeing this message because your Web browser does not support basic Web standards. Find out more about why this message is appearing and what you can do to make your experience on this site better.


ABOUT ARCHIVES
Advanced Search

Welcome   | My Account | E-mail Alerts | Access Rights | Sign In


  Vol. 130 No. 3, March 1976 TABLE OF CONTENTS
  Archives
  •  Online Features
  ORIGINAL ARTICLES
 This Article
 •References
 •Full text PDF
 •Send to a friend
 • Save in My Folder
 •Save to citation manager
 •Permissions
 Citing Articles
 •Citation map
 •Citing articles on HighWire
 •Contact me when this article is cited
 Related Content
 •Similar articles in this journal
 Social Bookmarking
  Add to CiteULike Add to Connotea Add to Del.icio.us Add to Digg Add to Reddit Add to Technorati Add to Twitter What's this?

Nursery Outbreak of Scalded-Skin Syndrome

Scarlatiniform Rash Due to Phage Group I Staphylococcus aureus

Howard S. Faden, MD; John P. Burke, MD; Lowell A. Glasgow, MD; James R. Everett III, MD; Linda Book, MD; Bill B. Wiley, PhD; Richard Warren, PhD; Liz Hirose

Am J Dis Child. 1976;130(3):265-268.


Abstract

• From Aug 6 to 14, 1973, scarlatiniform eruptions that were considered to be mild forms of the staphylococcal scalded-skin syndrome developed in four neonates. One infant had mild epidermal peeling. All had generalized, finely papular erythema that cleared rapidly after treatment with antibiotics.

Cultures from the umbilical stumps or anterior nares of three of the infants yielded colonies of group I Staphylococcus aureus, phage type 29/52/79/86/D11/81, that were able to produce epidermal exfoliation in suckling mice. These data indicate that the nursery outbreak was caused by phage group I staphylococci rather than group II organisms previously associated with staphylococcal scalded-skin syndrome. The demonstration that a group I Staphylococcus can produce exfoliative toxin suggests that the same mechanism for toxin production may exist for phage groups I and II staphylococci.

(Am J Dis Child 130:265-268, 1976)



Author Affiliations

This investigation was supported in part by grants AI 10217 and 3T01 A100039-15S1 from the National Institute of Allergy and Infectious Diseases.

From the departments of pediatrics (Drs Faden and Glasgow) and medicine (Drs Faden and Burke), University of Utah College of Medicine, and the Utah State Division of Health, Salt Lake City (Dr Everett); and the Bureau of Epidemiology, Center for Disease Control, Atlanta (Dr Everett).


Footnotes

Received for publication Dec 11, 1974; accepted April 10, 1975.

Reprint requests to Division of Infectious Diseases, University of Utah Medical Center, 50 N Medical Dr, Salt Lake City, UT 84132 (Dr Faden).



Add to CiteULike CiteULike   Add to Connotea Connotea   Add to Del.icio.us Del.icio.us   Add to Digg Digg   Add to Reddit Reddit   Add to Technorati Technorati   Add to Twitter Twitter     What's this?

THIS ARTICLE HAS BEEN CITED BY OTHER ARTICLES

Staphylococcal Scalded Skin Syndrome After Herniorrhaphy
ARTMAN et al.
Arch Pediatr Adolesc Med 1981;135:471-472.
ABSTRACT  

Nosocomial Scalded Skin Syndrome: Ritter's Disease Caused by Phage Group 3 Staphylococcus aureus
Florman and Holzman
Arch Pediatr Adolesc Med 1980;134:1043-1045.
ABSTRACT  

Toxic Epidermal Necrolysis (TEN; Ritter's Disease) and Staphylococcal Scalded Skin Syndrome (SSSS): A Description and Review
Koblenzer
CLIN PEDIATR 1976;15:724-730.
 





HOME | CURRENT ISSUE | PAST ISSUES | TOPIC COLLECTIONS | SUBMIT | SUBSCRIBE | HELP
CONDITIONS OF USE | PRIVACY POLICY | CONTACT US | SITE MAP
 
© 1976 American Medical Association. All Rights Reserved.